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In this study

after intraarticular injection of AdTSP-1

TSP-1 was detected in the synovial tissue of the treated joint and the progression of CIA was ameliorated. Furthermore

increased TGF_ expression

decreased production of IL-1_ and VEGF

and fewer blood vessels were observed in the synovial tissue from AdTSP-1–treated ankle joints. Our data indicate that TSP-1 gene transfer not only controlled pronounced inflammation and angiogenesis and suppressed synovial hypertrophy in the involved joint

but also had a strong protective effect on the cartilage and bone within the joint following the induction of CIA. It is likely that intraarticular injection of AdTSP-1 inhibited neoangiogenesis of developing inflammation and

furthermore

suppressed the established pannus. The detailed mechanismof TSP-1 gene therapy for the treatment of CIA

however

warrants further investigation. In conclusion

our results demonstrate that TSP-1 gene transfer can ameliorate the clinical course of rat CIA. These findings suggest that TSP-1 gene therapy may have therapeutic potential for the management of RA.TSP-1 gene transfer induced transforming growth factor _ (TGF_) expression and reduced infiltrating mononuclear cells in the synovial tissue of rats with collagen-induced arthritis. TGF_expression

concomitant with fewer mononuclear cells

was detected by immunohistochemistry in the synovial tissue from the AdTSP-1–treated ankle joint (A and B)

but not in that from the AdLacZ-treated ankle joint (C and D). Of note

TGF_expression was localized in the areas where infiltrating mononuclear cells were evident (A and B).
這個研究

AdTSP-1intraarticular注射後TSP-1用被對待了的接縫的滑液的組織被查出了

並且

CIA的進行被改善了。

而且

治療增加的TGF_表現

IL-1_和VEGF減少的生產

波及

更少的血管從AdTSP-1和#8211用滑液的組織被觀測了的;腳凳手。

我們的數據

在不僅僅被控制了的TSP-1遺傳基因引進表示與炎症新血管形成發音了的

有關的接縫壓制了滑液的肥大

不過

CIA的誘導之後

在接縫中再有著對軟骨和骨強的防護效果。

禁止AdTSP-1intraarticular注射展現的炎症的neoangiogenesis

而且

壓制了確立了的pannus的

快要有了。

然而

為了CIA的處理的TSP-1遺傳基因療法詳細的機械裝置保證新的調查。

作為結論

我們的結果

表示能TSP-1遺傳基因引進改善nezumiCIA臨床流逝。

這些的調查結果

說不定TSP-1遺傳基因療法有RA的管理的治療法的可能性表示的。

增生因子_一邊改變(TGF_)表現一邊被引起

骨膠原用伴隨被引起了的關節炎的nezumi的滑液的組織一邊滲透到單核細胞一邊被壓倒了的TSP-1遺傳基因引進。

更少的單核細胞並立的TGF_表現從根據根據在滑液的組織的免疫組織學從AdTSP-1和#8211被查出了的; AdLacZ被對待了的腳凳手(C和D)治療了腳凳手(A和B)

不過

因此不是應該治療了這樣的。

注意

TGF_表現滲透單核細胞明白的領域(A和B)被局部化做了。

�CIA的誘導之後

在接縫中再有著對軟骨和骨強的防護效果。

禁止AdTSP-1intraarticular注射展現的炎症的neoangiogenesis

而且

壓制了確立了的pannus的

快要有了。

然而

為了CIA的處理的TSP-1遺傳基因療法詳細的機械裝置保證新的調查。

作為結論

我們的結果

表示能TSP-1遺傳基因引進改善nezumiCIA臨床流逝。

這些的調查結果

說不定TSP-1遺傳基因療法有RA的管理的治療法的可能性表示的。

增生因子_一邊改變(TGF_)表現一邊被引起

骨

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